It has been 21 months of social upheaval and weirdness since the gift of love from the NIH and the Wuhan Institute of Virology.  In that time period, we have seen a widespread loss of faith in our government health apparatus based on contradictory, illogical, and frankly nefarious behavior.

I’ve divided this article into two parts:

Part number one was intended to be published on 18 November, but frankly, for a variety of reasons, including sloth, I didn’t get around to it.

Part two is being written today. I’m doing this in part because of new developments that are significant, and also as a bit of “I told you so”.

Part one: November 18

Overall U.S. Covid “cases” are falling, particularly in the South, while some isolated northern states are having surges.  We have seen this seasonal pattern before, with the arrival of the outdoor recreation season in some climates, while others seek shelter as temperatures drop.

If you look at recent data, the percentage of hospitalizations and death, as a percentage of the total “cases” has also been falling. This is likely due to multiple factors, including vaccination, increasing natural immunity, and better care particularly in the ICUs.  It may also be due in part to the more benign characteristics of the current “delta” variant which is now dominant in the population.

A virus can be thought of as a small biological machine.  They are not considered as living entities, but more as mechanisms.  The spread and dominance of variants tend to occur when mutations cause a particular set of useful characteristics:

1.  The virus evolves to be more infectious.  This could mean that smaller numbers of viruses are needed to institute an infection, and/or the virus changes characteristics to survive better between hosts.  The virus can also evolve to create symptoms in the host that facilitate spread, such as coughing or diarrhea.

2.  The virus becomes less lethal.  These little mechanisms derive no benefit from killing their host.  Arguably the virus can spread more rapidly if the symptoms are mild, and the host remains in contact with others.  Decreasing lethality will also be caused by an increase in acquired immunity in the population (and perhaps their offspring).  In other words, we and the virus evolve so that we can coexist.

Other than the outlier viruses SARS CoV, MERS Co-V, and SARS-CoV2, there have been four coronavirus types that typically circulate in the population and cause respiratory infections.  These typically cause mild to moderate symptoms though they too can be the cause of pneumonia, and ultimately death, in vulnerable populations.    They are estimated to be responsible for 10 to 30% of viral respiratory infections.

Now here’s the thing. Because they are generally benign, and there has been no therapy identified specifically for these viruses, we generally do not test for them.  And we certainly do not isolate or vaccinate. But we do for Covid.  And at some point, that is going to have to change.

Coronaviruses in general have been endemic in the human population for thousands and thousands of years.  Acquired immunity to them tends to be “relative” in that people tend to be reinfected multiple times in their lives, but typically with modest symptoms.

That may explain the performance of the current vaccines.  At this point, they do not seem to prevent infection and spread but do seem to blunt the severity of the illness that results (though this effect is also waning).  The point is, that SARS-CoV2, will likely become an endemic virus, and any expectation that you will not be reinfected at some point is probably unrealistic.  At some point, we will need to normalize our behavior towards this infection and cease with all the isolation and dramatics.

There is some hope.  As much as I despise Pharma, and their actions regarding hydroxychloroquine and Ivermectin, Merck, and now Pfizer, have introduced oral medications for Covid.  Happily, these drugs are patented, so the pharmaceutical companies will make a lot of money and so happily promote their use.

 Pfizer claims that their medication Paclovid, reduced hospitalization by 89% and death by 100% in their clinical trials.  If this bears out, it would definitely be a significant development, with the potential to change both the epidemiology and psychology of this illness.  Merck claims that their new medication Molnupiravir, was roughly 50/100% effective using the same criteria.

In case you wondered, in several meta-analyses, Ivermectin is about 65% for early treatment, with similar numbers for late treatment. It’s about 87% effective for prophylaxis.

I wonder, given these new medications, and the growing sense that the vaccines seem both relatively risky and ineffective, that Pharma will deemphasize the “jabs” for their new therapeutics.  This may be especially true now that monoclonal antibody cocktails given subcutaneously, have recently been shown to have potential as COVID-19 prophylaxis for as long as eight months.

Part two:

So that was written on November 15-17. It is now 2 weeks later and there have been significant developments since.

The number one development would be the continual deterioration in vaccine performance. We are now seeing significant numbers of fully vaccinated people admitted to ICUs, in some cases never discharged. Both director Walinsky, and Dr. Fauci have been forced to acknowledge this. Their answer: inject patients with even more spike protein mRNA.

There is an abstract published in the Journal Circulation that studied the body’s release of certain biomarkers predictive of cardiac complications after receiving the mRNA vaccine. It concluded:  “that the mRNA vacs dramatically increase inflammation on the endothelium and T cell infiltration of cardiac muscle and may account for the observations of increased thrombosis, cardiomyopathy, and other vascular events following vaccination.”

Now, remember back in 1998. A paper was published by one study group investigating the effect of hormone replacement therapy for postmenopausal females on the heart. It concluded surprisingly, that cardiac outcomes were worse in females who were receiving HRT. In an instant, HRT ceased as an option for women.

Let’s see whether this Circulation article has a similar effect on these vaccines (I doubt it).

Poor Merck. They spent the last year impugning their old drug Ivermectin for the treatment of Covid 19. Shockingly they then released their new oral therapeutic, Molnupiravir which they initially reported as having a 50% reduction in illness and death when used treatment of SARs-CoV2. This made Dr. Fauci very excited.

Fun fact: Molnupiravir works by inserting errors into the viral genetic code. Apparently, during the trials, subjects were required to refrain from heterosexual sexual intercourse, and in the case of females, be using birth control. What could go wrong?

Earlier this week they downgraded that effectiveness number to roughly 30%. This is a problem when Pfizer is reporting 89% effectiveness of their new oral medication Paclovid. If this data bears out I would imagine Molnupiravir will be a bust. Karma is a bitch.

Just so you know, the geniuses in our government have already arranged to buy $1.7 billion worth of this apparent loser. Oops.

  By the way, the much more effective Pfizer drug uses a similar mechanism of action to… You guessed it… Ivermectin.

And now we have the Omicron variant emerging from South Africa. This apparently has the World Health Organization in a tizzy, which the press is dutifully reporting. This variant is said to carry multiple mutations including changes to the spike protein (you know, where the vaccines work). It is said to be more infectious than Delta, but there are indications that symptoms are generally mild. There is obviously great hand wringing that it will be vaccine-resistant (like delta already is?).

Maybe they can lock us down again this Christmas.

To close this up, let me review the facts we need to remember to avoid being manipulated by the hype.

1. SARs-CoV2 is a respiratory virus, much less severe than SARs1, and over time, roughly as deadly as a bad influenza season.
2. We are being pushed to take multiple doses of hastily developed, still investigational monovalent vaccines. We are vaccinating during a pandemic which generally a bad idea. The vaccines over time do not really appear to be very effective.
3. The new variant is  likely doing what viruses are supposed to do; evolving to become more infectious but less virulent. It is frightening to the medical establishment, because its spike protein mutations threaten to completely discredit their entrenched “vaccine only “policies.
4. Given the large percentage the population with pre-existing Coronavirus exposure, and ongoing SARs-CoV2 infections, there is likely a high percentage of the population with broad-based immunity, even to new variants.
5. There are effective IV and now oral medications. Some are repurposed, but there is a new oral med they may actually allow us to use to treat this infection when it occurs.

I apologize for the length of this. I confess that writing these articles helps me to disperse the toxic degree of cognitive dissonance I experience when ingesting Covid information from the government and the media.

I very much appreciate your interest, your shares, and your comments.

Header image: Deer Camp near Sullivan Falls (Fujifilm X-T10, XF 18-55mm f2.8-4)

The forests in my neighborhood are dying.  Well not maybe the whole ecosystem, but sometimes it seems that in that at this time in history, there is an unprecedented onslaught against the eastern forest.

But it’s not true.  Such events have happened before. 

The American chestnut was arguably the most important tree species in the eastern forests of the 19^th century.  It was a large tree, at full maturity as tall as 120 ft., with the base of some tree 5 to 6 feet in diameter.  Its wood was light, yet strong and rot proof; useful in many functions including railroad ties, and house framing.  Its yearly crop of mast or nuts were an important food source for multiple species, including our own.   

In the early years of the 20^th century, a fungal pathogen known as the chestnut blight, arrived in the city of New York riding along on A shipment of Asian chestnut trees.  This blight quickly spread, and in 30 years, wiped out millions of chestnut trees throughout the east.  This extraordinary organism, many times serving as a node tree in what we know to be our interconnected forests, became virtually extinct.

If you walk along the trails around our community, you will periodically observe a cluster os sapling sized trunks interspersed with dead or dying snags. These will bear the distinctive serrated leaves of the American chestnut. There may be long strands of catkins in the spring, and a few nuts in the fall. These are the withered descendants of their once magnificent species. Each sapling will live a few years until they are girdled by the fungus, The roots, which are unaffected, will desperately send up more shoots. Thus they have existed for perhaps a century.

  A few mature trees still survive, either under the protection of humans in horticultural collections, or isolated in the backcountry.

  I have stood beside one such tree that exists deep in a draw on the west rim of Pennsylvania’s Grand Canyon.  There it has escaped, for now, the blight that has wiped out its relatives.  As I stood there alone, under its broad canopy, I was transfixed.

 There are currently efforts To revive the species using several techniques, including backcrossing the vulnerable Americans species with its resistant Asian cousins. Still, the loss of this magnificent species leaves me wistful in that I never was able to witness the beauty of the so-called chestnut-oak forest that was so much a part of our region.

Then there was Dutch elm disease, This was another Asian fungus that arrived by way of the Netherlands in the 1920s, and devastated our majestic American Elms. Some Elms still survive.

There certainly been other threats to forest members over the years. Our oak trees are constantly under attack by another Asian immigrant, the gypsy moth, which can cause multiple defoliations over time, killing even robustly healthy oak trees. We lost two magnificent roughly 150-year-old specimens several years ago due to an outbreak.

Our magnificent hemlocks, Pennsylvania’s state tree, is at risk due to the Hemlock Wooly Algedid, another imported pest.

Yet in both cases, the species are persevering.

Now over 100 years after the blight was introduced, we are once again faced with a similar situation.   

The ash tree is in a way, an unheralded species of tree common in Pennsylvania’s forests.  Its wood is light but strong, rather famously used to create the baseball bats used by major league baseball over the years.  Yet it’s an unassuming tree, without any flashy blooms, or plentiful fruits or nuts to remind us of its presence.

I fact I looked for images of ash trees in my archives. I found images of oak, maples and even spindly chestnuts. But none sadly, of the, now threatened tree.

Unfortunately, At least as a member of our forest community, It’s about to follow the chestnut tree into oblivion.

Again it is an Asian immigrant, this time an insect, The Emerald Ash Borer.  First found in the upper Midwest In the early two thousand, It is spread quickly Throughout the Eastern United States.  First noted in Pennsylvania and 2007. It spread quickly on the wing. It is virtually a 100% lethal, once it infests even healthy ash trees. 

We began to notice the signs last summer. Along the dirt road that leads to my home, there are at least 50 Green ash trees many of them quite mature. Last September their bark began to deteriorate as though someone had taken a giant sander to them.  This is called “blonding”, and is caused by peeling off the bark.   Soon we could see the characteristic “D “shaped holes in the bark caused by the insects.

 This spring, none of them have foliage. In fact, I have yet to find  an ash tree that has any sign of life

It is possible to save individual trees. I have none on my property, but if I did, they could be injected every couple of years with a pesticide that will protect them from the parasite. It is expensive, and as I understand it would need to go on for the foreseeable future. Sadly, it is the only way and is obviously not practical for the trees of our woodlands.

Science tells us, that our forests are complex communities, with fungi interconnecting the trees of many species. Trees it turns out can use these networks to communicate chemically.  They can warn each other of danger such as an influx of parasites., They will even share carbon and other nutrients when necessary.  Thus the forest behaves as a family of organisms, working together.

With this newest onslaught that will likely wipe out a species, I wonder:

Do the Oaks mourn?  

Every once in a while, perhaps while out shopping, or at a restaurant, I will encounter an old friend, whose life path has drifted out of my particular orbit. After spending some time catching up, I will be reminded of how much I enjoy their company, and will wonder why we didn’t make more of an effort to keep in touch.

In the last several weeks, I feel it had that sort of an experience with a lens. In particular, the Fujinon XF 60 mm f2.4.

This was one of the 3 original prime lenses introduced with the X Pro 1 in 2012. At the time, it was lauded by some is the sharpest of the trio (the 18 mm f 2.8, the 35mm F1.4, were the other 2 lenses).

I remember my initial impressions were that this was a bitingly sharp lens, noticeably more so than my Nikon primes. In fact, the quality of all of these original lenses probably helped pull me from using my Nikon gear, to shooting mainly with Fuji.

But just like with life, newer things come along, in this case, lenses that are faster and newer, and distract me from an old companion. After a time the 60 mm became lost in the back of my photography cabinet.

Several weeks ago while on my way out the door to go hiking, I picked up camera bag with my XT-10 body, which I noticed had no lens mounted on it. I opened a cabinet and on a whim, reached blindly to the back of the shelf, determined to utilize the first Fujifilm lens I grabbed. This turned out to be the 60 mm, which I have basically ignored for many years. I mounted it on the camera and have been shooting ever since.

To remind you, this is a macro lens, the first in the Fujifilm “XF” line. It is fairly diminutive, compared to for instance the 56 mm f.12. It’s nicely built, with not only a fairly deeply recessed small front element but then a rather large metal lens hood. All of this makes it quite resistant to lens flare as the sun’s rays rarely reach the front element.

Though the lens aperture is tighter than many of the other Fujifilm primes, the lens is quite sharp wide open at F2.4. For those of us that focus on landscape imaging, the modest deficit in light gathering is really not much of an impediment when shooting in the daylight hours. This plus its small size may make it preferable to the newer 56mm for shooting while hiking. If only it had stabilization…

On an APS sensor, the lens has a 90 mm equivalent field of view, which makes it ideal for the sort of late summer early fall photography, available now as it allows you to isolate the sporadic displays of autumnal color beginning to erupt at this time of year. It’s also wonderful to have its macro capabilities for close-ups of wildflowers and the other fall forest offerings.

And boy is it sharp! From f2.8 and above, the lens has incredible definition. Out of focus elements look lovely at f2.4-3.5. Mounted on the XT-10 it balances very nicely, though it is a bit long with the lens hood installed. Still an all, it’s very compact package.

One downside of this optic for outdoor use is that given its magnification, it is prone to artifacts from camera shake. I find that using my steel Gitzo monopod, and good shooting technique, I can get acceptably sharp results without a tripod. You just have to be careful.

I did a search on eBay and found there are a lot of these lenses for sale. I suspect that it is a common lens for Fujifilm users to unload. There is a wide range of price’s but it looks like you could acquire a nice used one for between $US200- $400. There is also a broad range of prices for new lenses of between $US300-$650.

Given my experience, I think I’m going to put this lens back into circulation for outdoor shooting, relying more on the 56 mm f1.2 for street, and indoor work.

If you have one in the back of your cabinet, pull it out. I suspect he will find it is indeed like an old friend.

 

 

 

 

There is a different and wonderful culture in the Adirondacks. It is one where outdoor activities feature prominently.  Events such as the Wooden Ski Race, and Full Moon Madness that I have described elsewhere on this site, as well as the Saranac Winter Festival, all display a physicality and an embrace of the season that is lacking for instance, in my Pennsylvania hometown.

On a recent trip to Lake Placid, I attended one of my favorites; namely the Broomball tournament, held at The Cottage, a waterside/ pub restaurant, that is part of the spectacular Mirror Lake Inn; a luxury resort hotel in the village.

The Cottage is the pub and informal dining venue for the resort. In summer can be very pleasant to paddle a kayak or canoe to the restaurant’s dock , and stop for a bite of lunch.

For much of the winter, however, with Mirror Lake firmly frozen, an area of the ice is plowed behind the restaurant, and an informal hockey rink is set up. Then in January, the restaurant sponsors a tournament for the somewhat arcane sport, which is apparently the winter version of softball for many people.

Broomball, is a sort of proto hockey. The rules are similar, but the equipment, at least in the case of the local variety is much less sophisticated. As I understand it was often played in tennis shoes, though there is actually a specialized shoe to wear which gives more friction to the icy surface. In the Lake Placid varient, the predominant “stick”, was an actual broom, with the bottom cut off about halfway up, then wrapped in duct tape. There is an actual broomball that is used, a little bigger than a softball.

While some of the teams, had actual matching hockey Jerseys (and apparently padding), most people played in heavy outdoor clothing sometimes with whimsical touches. Everyone plays with some sort of helmet. In this multisport town, I saw players sporting rock climber’s helmets, skiing helmets, and even hockey helmets. Though it was quite mild (high 20s F) this year, I have seen it proceed enthusiastically with temperatures in the minus teens.

Now to be sure, Broomball, apparently in Canada, and parts of the US, is a serious sport with a governing body, and specialized equipment, and national tournaments. Not so much in Lake Placid. Though obviously a lot of serious competitive athletics goes on in town, this one is not so intense. Like many activities in the North Country, my impression over the years from this event, it is that it is first and foremost an opportunity to socialize and be convivial on a dark cold evening in January. It is only secondarily a competition.

The large crowd gathers both in the restaurant, where windows allow the games to be followed, or down on the ice around a warming fire built for the occasion. As the festivities continue, the bar gets rowdy, but never unpleasantly so. Many of the older folks, who prudently no longer play, share great memories of their own time on the ice.

It is one of the small events, that over the year makes Lake Placid a truly unique placed to visit.